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Its primary pharmacological characterization was published in European Journal of Endocrinology (PMID 9849822), where the authors identified it as the first growth-hormone-secretagogue receptor (GHSR-1a) agonist achieving GH-release selectivity comparable to GHRH in animal models

doi: 10.1016/j.cmet.2013.07.002

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This disruption facilitates the release of cytochrome c into the cytoplasm, triggering caspase[29] activation and leading to apoptotic cell death

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